Explore by body system

Select an antibody to see which regions are affected and how commonly.

Jo-1
Anti-histidyl-tRNA synthetase
60–75% of ASyS cases
Lungs / ChestInterstitial lung disease (ILD)
Common
Proximal arm musclesShoulder & upper arm weakness
Common
Hands & fingersMechanic's hands · Raynaud's · Arthritis
Common
Proximal leg musclesThigh & hip weakness (myopathy)
Common
SystemicConstitutional fever & fatigue
Frequent
Throat / OesophagusDysphagia — difficulty swallowing
Occasional
The most common antisynthetase antibody and the most studied. Classical ASyS triad (myopathy, ILD, arthritis) is most consistently seen with Jo-1.
Prevalence in this antibody subtype: Common (>50%) Frequent (25–50%) Occasional (<25%)
Jo-1
60–75% of cases
Anti-histidyl-tRNA synthetase
Target: Histidyl-tRNA synthetase (HisRS)
Inflammatory myopathyOften the presenting feature
Common
Interstitial lung disease70–80% of Jo-1 patients
Common
Arthritis / arthralgia
Common
Mechanic's hands
Common
Raynaud's phenomenon
Frequent
Fever
Frequent
Dysphagia
Occasional
The most common antisynthetase antibody and the most studied. Classical ASyS triad (myopathy, ILD, arthritis) is most consistently seen with Jo-1.
PL-7ILD-predominant
5–10% of cases
Anti-threonyl-tRNA synthetase
Target: Threonyl-tRNA synthetase (ThrRS)
Interstitial lung diseaseOften severe; may precede myopathy
Common
Inflammatory myopathyTypically milder than Jo-1
Frequent
Arthritis / arthralgia
Frequent
Mechanic's hands
Frequent
Raynaud's phenomenon
Frequent
Fever
Occasional
ILD is more dominant than myopathy. Patients may present to respiratory physicians before a rheumatology diagnosis is made.
PL-12ILD-predominant
5–10% of cases
Anti-alanyl-tRNA synthetase
Target: Alanyl-tRNA synthetase (AlaRS)
Interstitial lung diseaseOften severe and progressive
Common
Raynaud's phenomenon
Common
Mechanic's hands
Frequent
Inflammatory myopathyLess prominent than other subtypes
Occasional
Arthritis / arthralgia
Occasional
Fever
Occasional
Strongly ILD-predominant. Myopathy may be subclinical or absent. Often initially misdiagnosed as idiopathic pulmonary fibrosis.
EJ
3–5% of cases
Anti-glycyl-tRNA synthetase
Target: Glycyl-tRNA synthetase (GlyRS)
Interstitial lung disease
Common
Inflammatory myopathy
Common
Arthritis / arthralgia
Frequent
Mechanic's hands
Frequent
Raynaud's phenomenon
Occasional
Fever
Occasional
Phenotype similar to Jo-1 with both myopathy and ILD prominent. Limited data due to rarity.
OJILD-predominant
2–4% of cases
Anti-isoleucyl-tRNA synthetase
Target: Isoleucyl-tRNA synthetase (IleRS)
Interstitial lung diseaseFrequently severe
Common
Inflammatory myopathyCan be severe
Common
Mechanic's hands
Frequent
Raynaud's phenomenon
Occasional
Arthritis / arthralgia
Occasional
Both ILD and myopathy can be severe. One of the rarer subtypes with limited prospective data.
KSILD-predominant
2–3% of cases
Anti-asparaginyl-tRNA synthetase
Target: Asparaginyl-tRNA synthetase (AsnRS)
Interstitial lung diseasePredominant feature
Common
Raynaud's phenomenon
Frequent
Fever
Frequent
Inflammatory myopathyOften subclinical
Occasional
Arthritis / arthralgia
Occasional
Mechanic's hands
Occasional
Strongly ILD-predominant with frequent Raynaud's and fever. Myopathy is often mild or absent entirely.
Ha / YRS
<1% of cases
Anti-tyrosyl-tRNA synthetase
Target: Tyrosyl-tRNA synthetase (TyrRS)
Interstitial lung disease
Common
Inflammatory myopathy
Frequent
Mechanic's hands
Frequent
Arthritis / arthralgia
Frequent
Raynaud's phenomenon
Occasional
Very rare. Clinical data are limited to case reports and small series. Phenotype appears to resemble Jo-1 ASyS.
Zo
<1% of cases
Anti-phenylalanyl-tRNA synthetase
Target: Phenylalanyl-tRNA synthetase (PheRS)
Interstitial lung disease
Common
Inflammatory myopathy
Frequent
Mechanic's hands
Occasional
Arthritis / arthralgia
Occasional
Extremely rare. Fewer than 20 cases described in the literature. Clinical significance and full phenotypic spectrum remain under investigation.

Quick comparison

AntibodyCasesMyopathyILDArthritisMechanic's handsRaynaud's
Jo-160–75%
PL-75–10%
PL-125–10%
EJ3–5%
OJ2–4%
KS2–3%
Ha / YRS<1%
Zo<1%

Prevalence estimates are approximations based on published literature. Individual patient presentations vary considerably.

Sources & further reading

  • Connors GR et al. Interstitial lung disease associated with the idiopathic inflammatory myopathies. Eur Respir Rev. 2010.
  • Lega JC et al. The clinical phenotype associated with myositis-specific and associated autoantibodies. Semin Arthritis Rheum. 2014.
  • Hamaguchi Y et al. Clinical features of anti-aminoacyl-tRNA synthetase antibody-positive patients. Mod Rheumatol. 2013.
  • Mahler M et al. Autoantibodies in antisynthetase syndrome. Autoimmun Rev. 2021.
  • Gutsche M et al. Connective tissue disease-associated interstitial lung disease. Semin Respir Crit Care Med. 2012.